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Rod and cone degeneration in the rd mouse is p53 independent

J Wu1, J Trogadis, R Bremner

  • 1Vision Science Research Program, Cellular and Molecular Division, Toronto Western Research Institute, University Health Network, Toronto, Canada.

Molecular Vision
|May 10, 2001
PubMed
Abstract

Insights

The tumor suppressor p53 (p53) is not essential for photoreceptor or inner nuclear layer cell death in rd mice, a model for retinitis pigmentosa. This study clarifies p53

Area of Science:

  • Ophthalmology
  • Genetics
  • Cell Biology

Background:

  • Retinitis pigmentosa (RP) is a group of inherited retinal diseases.
  • Photoreceptor degeneration is a hallmark of RP.
  • The role of p53 in retinal apoptosis is not fully understood.

Purpose of the Study:

  • To investigate the necessity of p53 for photoreceptor (rod and cone) death in rd mice.
  • To determine if p53 is required for inner nuclear layer (INL) cell degeneration in developing retinas.

Main Methods:

  • Apoptosis was assessed using TUNEL staining in p53+/+ and p53-/- rd mouse retinas at various postnatal days.
  • Cone photoreceptor survival was quantified by counting peanut agglutinin (PNA)-positive cells in whole mount retinas.

Main Results:

  • The absence of p53 did not alter the rate of rod and INL cell death.
  • Cone photoreceptor survival remained unaffected in p53 knockout rd mice.

Conclusions:

  • p53 is not essential for the degeneration of rod or cone photoreceptors in the rd mouse model.
  • p53 does not play a critical role in the elimination of INL cells during late retinal development.

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