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Related Experiment Videos

Alleviating mortality associated with a vitamin E-selenium deficiency by dietary ascorbic acid.

E T Moran, H C Carlson, R G Brown

    Poultry Science
    |January 11, 1975
    PubMed
    Summary

    Adding ascorbic acid to duck feed significantly lowered mortality in a study. This suggests that vitamin C supplementation can help prevent serious health issues in ducks with specific dietary deficiencies.

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    Area of Science:

    • Animal Nutrition
    • Biochemistry
    • Avian Pathology

    Background:

    • Growing ducks fed practical rations often develop deficiencies in essential nutrients like vitamin E and selenium.
    • These deficiencies can lead to significant mortality and specific health issues, including myopathies and vascular faults.
    • The role of endogenous ascorbic acid (vitamin C) synthesis in ducks under such conditions is not fully understood.

    Purpose of the Study:

    • To investigate the effect of supplemental ascorbic acid on mortality in growing ducks fed a diet deficient in vitamin E and selenium.
    • To explore the potential role of impaired de novo ascorbate synthesis in the observed pathologies.

    Main Methods:

    • Growing ducks were fed a practical ration deficient in vitamin E and selenium.
    • Ascorbic acid was added to the ration for a subset of the ducks.

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  • Mortality rates and the presence of myopathies and vascular faults were monitored.
  • Main Results:

    • Supplementation with ascorbic acid substantially reduced mortality in the ducks.
    • Myopathies were observed in the ducks, but vascular faults were absent.
    • The findings suggest a link between the dietary deficiencies and impaired ascorbate synthesis.

    Conclusions:

    • Ascorbic acid supplementation is effective in reducing mortality in growing ducks with vitamin E and selenium deficiencies.
    • The study implies that a secondary inadequacy of ascorbate synthesis may contribute to the observed pathologies and mortality.
    • Alleviating this induced ascorbate inadequacy is likely the primary mechanism behind the reduced death rate.