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Tamoxifen citrate induced sperm shape abnormalities in the in vivo mouse

S Padmalatha Rai1, K K Vijayalaxmi

  • 1Department of Biotechnology, Kuvempu University, Jnana Sahyadri, 577451, Shimoga, India. root@shikuvem-kar.nic.in

Mutation Research
|May 30, 2001
PubMed

Insights

Tamoxifen citrate, an anticancer drug, showed genotoxic potential in mice. The study found statistically significant sperm abnormalities, indicating the drug

Area of Science:

  • Pharmacology
  • Toxicology
  • Genetics

Background:

  • Tamoxifen citrate is a widely used anticancer drug.
  • Assessing the genotoxic potential of pharmaceuticals is crucial for patient safety.

Purpose of the Study:

  • To evaluate the genotoxic effects of tamoxifen citrate.
  • To determine the relationship between tamoxifen citrate dosage and sperm abnormalities in vivo.

Main Methods:

  • In vivo mouse model (Swiss albino mice).
  • Sperm abnormality assay conducted after 5 weeks.
  • Repeated administration of tamoxifen citrate at doses of 0.1, 0.2, 0.4, and 0.8 mg/kg b.w. per day for 10 days.

Main Results:

  • Statistically significant increases in sperm abnormalities were observed.
  • The genotoxic effects were dose-dependent, with higher doses causing more abnormalities.

Conclusions:

  • Tamoxifen citrate exhibits genotoxic potential.
  • The sperm abnormality assay is a sensitive method for detecting drug-induced genotoxicity.
  • Further research into the long-term effects and mechanisms of tamoxifen citrate genotoxicity is warranted.

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