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Schizophrenia and liver dysfunction.

J Krøll1

  • 1Dept of Clinical Chemistry, Blekinge County Hospital, Karlskrona, Sweden. krollj@danbbs.dk

Medical Hypotheses
|June 5, 2001
PubMed
Summary

Non-hepatocellular liver dysfunction from portal-systemic shunts may predispose individuals to schizophrenia. Further research using Doppler sonography is needed to determine the frequency of these shunts in schizophrenia patients.

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Area of Science:

  • Neuroscience
  • Hepatology
  • Psychiatry

Background:

  • Non-hepatocellular liver dysfunction is increasingly recognized.
  • Portal-systemic shunts can lead to significant physiological alterations.
  • Schizophrenia pathogenesis remains incompletely understood.

Purpose of the Study:

  • To explore the potential role of liver dysfunction due to portal-systemic shunts in schizophrenia development.
  • To investigate shared biochemical and biological markers between schizophrenia and portal-systemic shunting.
  • To propose a method for quantifying the prevalence of portal-systemic shunts in schizophrenia.

Main Methods:

  • Review of existing literature on liver disease and schizophrenia.
  • Analysis of shared autoptic and biochemical findings.
  • Proposal for utilizing non-invasive Doppler-sonographic techniques.

Main Results:

  • Evidence suggests a link between liver dysfunction (specifically portal-systemic shunts) and schizophrenia.
  • Numerous biochemical and biological abnormalities are common to both conditions.
  • The exact frequency of portal-systemic shunts in schizophrenia is currently unknown.

Conclusions:

  • Portal-systemic shunts causing non-hepatocellular liver dysfunction may be a significant predisposing factor for schizophrenia.
  • Further investigation into the prevalence of these shunts is warranted.
  • Doppler sonography offers a promising avenue for future research in this area.

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