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Enterobacter species in a pediatric hospital: horizontal transfer or selection in individual patients?

P de Man1, E van Der Veeke, M Leemreijze

  • 1Department of Medical Microbiology and Infectious Diseases, Erasmus University Medical Center, Rotterdam, The Netherlands. peter_de_man@franciscus.nl

Insights

Cross-transmission, not patient gut flora, primarily drives Enterobacter colonization in children. This study highlights hospital-acquired infections and the spread of resistant bacteria among young patients.

Area of Science:

  • Microbiology
  • Infectious Diseases
  • Pediatrics

Background:

  • Enterobacter species are significant nosocomial pathogens, particularly in pediatric settings.
  • The predominant source of Enterobacter colonization has been presumed to be endogenous gut flora.
  • Understanding transmission dynamics is crucial for infection control in hospitals.

Purpose of the Study:

  • To investigate the longitudinal epidemiology of Enterobacter colonization in a pediatric hospital.
  • To determine the primary sources and transmission routes of Enterobacter strains in hospitalized children.
  • To assess the prevalence of cross-transmission versus endogenous selection of Enterobacter.

Main Methods:

  • Longitudinal study of 171 children across 15 wards over two years.
  • Isolation and characterization of 287 Enterobacter species.
  • Molecular strain typing using random amplified polymorphic DNA (RAPD) and pulsed-field gel electrophoresis (PFGE).

Main Results:

  • Identified 97 distinct Enterobacter DNA types and 199 colonization events.
  • A predominant clone accounted for 111 isolates (39%) from 62 children.
  • Cross-transmission was identified as a major route, with 34% of children colonized by strains found in 2-4 other patients, contradicting endogenous selection theories.

Conclusions:

  • Cross-transmission plays a more significant role in Enterobacter colonization among hospitalized children than previously thought.
  • Hospital infection control strategies should focus on preventing the spread of Enterobacter between patients.
  • Findings challenge the assumption that resistant Enterobacter strains are primarily selected from a patient's own microbiota.

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