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Inhibitory effects of thymoquinone against 20-methylcholanthrene-induced fibrosarcoma tumorigenesis

O A Badary1, A M Gamal El-Din

  • 1Department of Pharmacology and Toxicology, College of Pharmacy, Al-Azhar University, Nasr City, Cairo, Egypt.

Insights

Thymoquinone (TQ) shows potential as a chemopreventive agent against fibrosarcoma. It inhibited tumor growth and survival in mice and cancer cells, possibly via antioxidant and DNA synthesis interference.

Area of Science:

  • Pharmacology
  • Oncology
  • Biochemistry

Background:

  • 20-methylcholanthrene (MC) induces fibrosarcoma in mice.
  • Thymoquinone (TQ) is a key compound in Nigella sativa seeds.
  • Investigating TQ's chemopreventive potential against MC-induced fibrosarcoma.

Purpose of the Study:

  • To evaluate the antitumor effects of TQ against MC-induced fibrosarcoma.
  • To explore the underlying mechanisms of TQ's action.
  • To assess TQ's impact on tumor incidence, burden, mortality, and specific biochemical markers.

Main Methods:

  • In vivo studies: TQ administration before and after MC treatment in mice.
  • In vitro studies: TQ treatment of fibrosarcoma cells.
  • Biochemical analysis: Measurement of lipid peroxides, glutathione (GSH), glutathione S-transferase (GST), and quinone reductase (QR) in liver tissues.

Main Results:

  • TQ significantly inhibited tumor incidence (43%) and burden (34%) in MC-treated mice.
  • TQ delayed tumor onset and reduced mortality, while restoring antioxidant markers (GSH, GST, QR) and reducing lipid peroxides.
  • In vitro, TQ inhibited fibrosarcoma cell survival (IC50 = 15 microM) and DNA synthesis.

Conclusions:

  • Thymoquinone demonstrates significant chemopreventive effects against MC-induced fibrosarcoma.
  • TQ's mechanisms involve antioxidant activity, interference with DNA synthesis, and enhanced detoxification.
  • TQ holds promise as a chemopreventive agent for fibrosarcoma.

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