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Survival of nontypeable Haemophilus influenzae in macrophages

J E Craig1, A Cliffe, K Garnett

  • 1University of Manchester, School of Biological Sciences, 1.800, Stopford Building, Manchester M13 9PT, UK.

FEMS Microbiology Letters
|September 15, 2001
PubMed

Insights

Nontypeable Haemophilus influenzae, NT477, survives longer within mouse macrophages than strain Rd. Most clinical isolates also resist macrophage killing, indicating varied survival capabilities in nontypeable strains.

Area of Science:

  • Microbiology
  • Immunology
  • Bacterial Pathogenesis

Background:

  • Nontypeable Haemophilus influenzae (NTHi) is a significant human pathogen, often causing respiratory tract infections.
  • The ability of NTHi to evade host immune responses, particularly macrophage clearance, is crucial for its pathogenesis.
  • Understanding NTHi survival mechanisms within macrophages can inform therapeutic strategies.

Purpose of the Study:

  • To investigate the intracellular survival of a specific NTHi strain (NT477) in a mouse macrophage cell line.
  • To compare the survival of NT477 with an avirulent control strain (Rd).
  • To determine the prevalence of macrophage survival traits among clinical NTHi isolates from otitis media patients.

Main Methods:

  • Phagocytosis assays using J774 mouse macrophage-like cells and bacterial strains NT477 and Rd.
  • Enumeration of viable intracellular bacteria at various time points post-infection (24h and 72h).
  • Screening of 33 primary clinical NTHi isolates for intracellular survival in macrophages.

Main Results:

  • Nontypeable Haemophilus influenzae NT477 survived intracellularly in macrophages for up to 72 hours.
  • In contrast, the avirulent strain Rd was killed within 24 hours.
  • 82% of 33 clinical NTHi isolates demonstrated persistence within macrophages for at least 24 hours, with varying recovery levels.

Conclusions:

  • NTHi strain NT477 possesses unique attributes for prolonged intracellular survival in macrophages compared to strain Rd.
  • The capacity for macrophage survival is a common trait among clinical NTHi isolates from otitis media.
  • There is significant inter-strain variability in the ability of NTHi to resist macrophage-mediated killing.

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