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Relationship between bronchial responsiveness and clinical evolution in infants who wheeze: a four-year prospective

C Delacourt1, M R Benoist, S Waernessyckle

  • 1Laboratoire d'Explorations Fonctionnelles Respiratoires, Service de Pneumologie et Allergologie Pédiatriques, Hôpital des Enfants Malades, Paris, France.

Insights

Bronchial hyperresponsiveness in infants with recurrent wheezing does not predict persistent asthma. While hyperresponsiveness persists, it doesn't reliably identify children who will develop long-term asthma.

Area of Science:

  • Pediatric Pulmonology
  • Allergy and Immunology
  • Respiratory Medicine

Background:

  • Recurrent wheezing in early childhood is often transient.
  • The role of bronchial hyperresponsiveness in persistent childhood asthma is not well understood.
  • Predicting which infants with wheezing will develop persistent asthma is clinically important.

Purpose of the Study:

  • To investigate if initial or persistent bronchial hyperresponsiveness differentiates infants with transient wheezing from those who develop persistent asthma.
  • To assess the predictive value of bronchial hyperresponsiveness for asthma persistence in early childhood.

Main Methods:

  • Prospective study of 129 infants with recurrent wheezing (≥3 episodes before 2 years).
  • Clinical evaluations and methacholine challenges (measuring PD(15) PtcO(2)) every 6 months for 4 years, then annually.
  • Assessment of VmaxFRC and SRaw values.

Main Results:

  • Most children's wheezing resolved; 20% had persistent wheezing at 4 years.
  • Persistent wheezers showed lower initial VmaxFRC and higher SRaw at follow-up.
  • Bronchial hyperresponsiveness persisted in all children, but persistent wheezers had lower PD(15) PtcO(2) from 30 months; however, no reliable predictive cutoff was found.

Conclusions:

  • The initial degree of bronchial hyperresponsiveness in infants with wheezing does not predict later asthma persistence.
  • While bronchial hyperresponsiveness persists, its level at 30 months is not a reliable early predictor for subsequent clinical progression.
  • Current measures of bronchial hyperresponsiveness in infancy are insufficient to predict the persistence of asthma into later childhood.

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