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How to get from antiplatelet to antithrombotic treatment
1Boehringer Ingelheim GmbH, Binger Strasse 173, 55216 Ingelheim, Germany. eisert@ing.boehringer-ingelheim.com
Insights
Chronic antiplatelet therapy offers moderate protection against myocardial infarction and stroke. Novel antithrombotic strategies amplifying endogenous defense systems show greater efficacy than solely inhibiting platelets.
Area of Science:
- Cardiovascular Medicine
- Pharmacology
- Thrombosis Research
Background:
- Chronic antiplatelet therapy with aspirin and clopidogrel provides limited benefit in preventing myocardial infarction and stroke.
- Combining antiplatelet agents increases cardiac event protection but elevates bleeding risk.
- Potent platelet aggregation inhibition and fibrinogen receptor antagonists have not improved clinical outcomes and show increased mortality.
Purpose of the Study:
- To evaluate the efficacy of current antiplatelet therapies in chronic settings.
- To investigate the role of endogenous antithrombotic defense systems.
- To explore novel antithrombotic strategies beyond simple platelet inhibition.
Main Methods:
- Analysis of clinical trial data on antiplatelet agents.
- In vitro and ex vivo platelet aggregation tests.
- In vivo studies of thrombus formation and antithrombotic factor interaction.
- Evaluation of combined antithrombotic therapies.
Main Results:
- In vitro platelet aggregation tests do not adequately predict clinical outcomes for chronic antithrombotic therapy.
- Endogenous antithrombotic defense systems play a crucial role in regulating thrombus formation.
- Combined therapies amplifying endogenous systems demonstrate superior antithrombotic effects compared to platelet inhibition alone.
- In vivo studies highlight the importance of balancing multiple antithrombotic treatments.
Conclusions:
- Current antiplatelet therapy monitoring via platelet aggregation tests is insufficient for chronic treatment selection.
- Future antithrombotic strategies should focus on amplifying endogenous defense mechanisms alongside or instead of solely inhibiting platelets.
- A shift towards comprehensive antithrombotic approaches, including vessel wall-mediated effects, is necessary for improved clinical outcomes.
Abstract:
Despite undisputed clinical benefit of platelet inhibitors in an acute interventional setting, chronic antiplatelet treatment with aspirin and clopidogrel have shown a moderate reduction of approximately 18% in the prevention of myocardial infarction and stroke. More recent data on the effect of combining the two compounds leading to an irreversible blockade of cyclooxygenase as well as the adenosine diphosphate receptor showed increased protection from cardiac events in patients with unstable angina and no significant protection from stroke. This improvement did come with the price of a significant increase in major bleeding, requiring hospitalization or transfusion. Furthermore, the most potent inhibition of platelet aggregation by blocking the binding of fibrinogen to activated platelets did not further improve the clinical outcome. All major clinical investigations of the effect of chronic administration have been recently terminated owing to an overall increased mortality rate for the entire class of drugs of orally active fibrinogen receptor antagonists. This poses the question of whether measuring platelet aggregation in vitro and ex vivo can serve as an adequate surrogate parameter to select antithrombotic therapy, particularly in the chronic setting. When evaluating the importance of other endogenous antithrombotic defense systems, the test for inhibition of platelet aggregation must fail. The importance of the endogenous defense systems is easily demonstrated by in vivo studies of thrombus formation in smaller vessels. The rapid and well-balanced interaction of local prothrombotic as well as antithrombotic factors is key in keeping the process confined. By carefully shifting this balance with antiplatelet therapy in combination with therapy to amplify the endogenous antithrombotic defense systems, a far more pronounced overall antithrombotic therapeutic effect can be demonstrated. By employing more complex antithrombotic tests performed in vivo, this balance can be studied. These studies confirm the importance of the ratio of two independent antithrombotic treatments when given together, which was not reflected in in vitro tests but was found to correlate with clinical outcome data. The attention in selecting antithrombotic treatment is to be shifted from approaches only directed toward the inhibition of platelets to an antithrombotic treatment, which includes amplification of the local therapeutic effects often mediated through the vessel wall.