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Phase variation among major surface antigens of Mycoplasma penetrans

K Röske1, A Blanchard, I Chambaud

  • 1Institut für Medizinische Mikrobiologie und Hygiene, Universitätsklinikum Carl Gustav Carus, Technische Universität Dresden, D-01307 Dresden, Germany.

Infection and Immunity
|November 14, 2001
PubMed

Insights

Mycoplasma penetrans exhibits phase-variable lipid-associated membrane proteins (LAMPs) that change independently, potentially helping the bacteria evade host immunity. This variation was observed in clinical isolates and the type strain.

Area of Science:

  • Microbiology
  • Immunology
  • Molecular Biology

Background:

  • The role of Mycoplasma penetrans in human health is not fully understood.
  • Lipid-associated membrane proteins (LAMPs), including the P35 antigen, are key targets for serological detection but are known to be phase-variable.

Purpose of the Study:

  • To comparatively analyze LAMP expression patterns in different Mycoplasma penetrans strains.
  • To investigate the intraclonal variation and frequencies of LAMP expression.
  • To explore the potential role of LAMP variation in immune evasion.

Main Methods:

  • Comparative analysis of LAMP profiles using SDS-PAGE and immunoblotting.
  • Monitoring phase variation of specific LAMPs (P34A, P34B, P35, P38) using monoclonal antibodies.
  • DNA sequencing of the P38 antigen gene.
  • Analysis of patient serum antibodies against LAMPs.

Main Results:

  • Clinical isolates and the type strain of Mycoplasma penetrans displayed distinct LAMP repertoires.
  • Phase variation of LAMPs occurred independently at frequencies of 10^-2 to 10^-4 per cell per generation.
  • The P34B and P38 antigens were cell-surface exposed, and P38 shared sequence homology with P35.
  • Patient antibodies recognized a P36 antigen not present in the patient's isolate, suggesting in vivo phase variation.

Conclusions:

  • Mycoplasma penetrans exhibits significant, independent phase variation in its LAMPs.
  • These variable LAMPs are likely involved in evading the host immune system.
  • The findings contribute to understanding Mycoplasma penetrans pathogenicity and immune evasion strategies.

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