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Pathogenicity of mumps virus in the marmoset

Shizuko Saika1, Minoru Kidokoro, Tokitada Ohkawa

  • 1Chiba Serum Institute, Chiba, Japan. kaihatu@triton.ocn.ne.jp

Journal of Medical Virology
|December 19, 2001
PubMed

Insights

Wild-type mumps virus caused meningitis in marmosets, unlike the Jeryl Lynn vaccine strain. This study highlights the marmoset model for assessing mumps virus neurovirulence potential.

Area of Science:

  • Virology
  • Neuroscience
  • Primate Models

Background:

  • Mumps virus can cause central nervous system (CNS) complications, including meningitis.
  • Understanding the neurovirulence of different mumps virus strains is crucial for public health and vaccine development.

Purpose of the Study:

  • To compare the neurovirulence of the wild-type mumps virus Odate strain and the Jeryl Lynn vaccine strain in a marmoset model.
  • To investigate the potential of the marmoset model for evaluating mumps virus neurovirulence.

Main Methods:

  • Intravenous inoculation of marmosets with the wild-type Odate strain or the Jeryl Lynn vaccine strain.
  • Clinical observation and histopathological examination of marmosets for CNS and other organ involvement.
  • Quantification of viral multiplication in marmosets.

Main Results:

  • The wild-type Odate strain induced meningitis and other organ pathologies (nephritis, parotitis, pancreatitis, tonsillitis) in marmosets.
  • The Jeryl Lynn vaccine strain did not induce CNS histopathological changes and showed lower viral multiplication compared to the Odate strain.
  • This is the first report of meningitis induction in non-human primates via peripheral inoculation of wild-type mumps virus.

Conclusions:

  • The Odate strain exhibits significant neurovirulence in marmosets, causing meningitis and systemic illness.
  • The Jeryl Lynn strain demonstrates a markedly reduced neurovirulence profile in this model.
  • The marmoset model is suitable for evaluating the neurovirulence potential of mumps virus strains, including vaccine candidates.

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