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Human T-lymphocyte cytotoxicity and proliferation directed by a single chimeric TCRzeta /CD28 receptor

John Maher1, Renier J Brentjens, Gertrude Gunset

  • 1Department of Human Genetics/Medicine, Gene Transfer and Somatic Cell Engineering Laboratory, and Immunology Program, Memorial Sloan-Kettering Cancer Center, New York, NY 10021, USA.

Nature Biotechnology
|December 26, 2001
PubMed

Insights

Engineered T lymphocytes targeting prostate-specific membrane antigen (PSMA) with novel fusion receptors effectively kill cancer cells. These modified cells also proliferate and produce IL-2, enhancing adoptive immunotherapy potential.

Area of Science:

  • Immunology
  • Cancer Biology
  • Biotechnology

Background:

  • Current artificial receptors for T cell targeting have limitations.
  • Existing receptors provide only activation or co-stimulatory signals, restricting cellular functions.

Purpose of the Study:

  • To develop and evaluate fusion receptors for enhanced T cell-mediated cancer immunotherapy.
  • To investigate the efficacy of T cells engineered with combined TCR-zeta and CD28 signaling elements against prostate-specific membrane antigen (PSMA)-expressing tumors.

Main Methods:

  • Retroviral transduction of human primary T lymphocytes with fusion receptors targeting PSMA.
  • Assessment of tumor cell lysis, T cell proliferation, and cytokine production (IL-2) upon stimulation.

Main Results:

  • Engineered T cells effectively lysed PSMA-expressing tumor cells.
  • Transduced lymphocytes exhibited robust proliferation (over 2 logs in 3 weeks) and significant IL-2 production.
  • Amplified T cell populations maintained antigen-specific cytolytic activity.

Conclusions:

  • Fusion receptors combining TCR-zeta and CD28 signaling moieties effectively redirect and amplify human T cell responses.
  • This approach holds significant promise for adoptive immunotherapy, particularly for cancers with compromised antigen presentation or co-stimulatory molecule expression.

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