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The effect of IFNbeta-1b on the evolution of enhancing lesions in secondary progressive MS

P A Brex1, P D Molyneux, P Smiddy

  • 1NMR Research Unit, Institute of Neurology, University College London, UK.

Neurology
|January 5, 2002
PubMed
Abstract

Insights

Interferon beta-1b (IFNbeta-1b) reduces new multiple sclerosis (MS) lesions, especially smaller ones. However, IFNbeta-1b does not change the progression of established lesions in MS patients.

Area of Science:

  • Neuroimaging
  • Neurology
  • Pharmacology

Background:

  • Multiple sclerosis (MS) lesions typically become isointense on T1-weighted MRI post-enhancement.
  • Hypointense T1 lesions indicate more severe tissue damage.
  • Interferon beta (IFNbeta) is known to reduce new enhancing MS lesions and their enhancement duration.

Purpose of the Study:

  • To investigate if IFNbeta affects tissue damage severity in new MS lesions.
  • To determine if IFNbeta's effects on lesion damage are size-dependent.

Main Methods:

  • 125 patients with secondary progressive MS were randomized to IFNbeta-1b or placebo.
  • Monthly contrast-enhanced T1-weighted MRI scans were performed for 24 months.
  • The size and follow-up appearance of new enhancing lesions (months 1-6) were documented.

Main Results:

  • IFNbeta-1b significantly reduced the number of new enhancing lesions in the first 6 months.
  • The reduction was more pronounced for small (70% decrease) versus large (46% decrease) lesions.
  • Hypointense T1 lesions developed more frequently from large (25%) than small (9%) enhancing lesions, irrespective of treatment.

Conclusions:

  • IFNbeta-1b effectively decreases the number of new enhancing MS lesions, particularly smaller ones.
  • IFNbeta-1b treatment did not alter the subsequent development of hypointense T1 lesions once an enhancing lesion was established.

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