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Nailfold capillary abnormalities in patients with familial Mediterranean fever

A Dinç1, M Melikoğlu, C Korkmaz

  • 1Division of Rheumatology, Department of Medicine, Cerrahpasa Medical Faculty, University of Istanbul, Turkey.

Abstract

Insights

Nailfold capillary abnormalities, including increased loop tortuosity and enlargement, are present in patients with familial Mediterranean fever (FMF). These findings correlate with female sex and Raynaud

Area of Science:

  • Rheumatology
  • Vascular Biology
  • Genetics

Background:

  • Familial Mediterranean fever (FMF) is a genetic autoinflammatory disorder.
  • Microvascular changes may contribute to FMF pathophysiology.
  • Nailfold capillaroscopy is a non-invasive method to assess microcirculation.

Purpose of the Study:

  • To quantify the frequency and severity of nailfold capillary abnormalities in FMF patients.
  • To compare capillaroscopic findings in FMF patients with healthy controls and other rheumatic diseases.
  • To identify factors associated with capillaroscopic changes in FMF.

Main Methods:

  • Conventional capillaroscopy was used to evaluate nailfold capillaries in 67 FMF patients, 37 healthy controls, 19 SLE patients, and 8 PSS patients.
  • Capillary loops were assessed for avascular areas, tortuosity, enlargement, and extravasations.
  • Participants were surveyed for Raynaud's phenomenon (RP), and FMF patients and controls underwent blind examination.

Main Results:

  • FMF patients exhibited significantly increased capillary loop tortuosity and enlargement compared to healthy controls.
  • No significant difference in microhemorrhages was observed between FMF patients and controls.
  • Female sex and the presence of Raynaud's phenomenon were correlated with capillaroscopic abnormalities in FMF.

Conclusions:

  • Nailfold capillaroscopy reveals characteristic capillary abnormalities in patients with familial Mediterranean fever.
  • These microvascular changes may be relevant to the clinical manifestations of FMF.
  • Further research is warranted to elucidate the role of these abnormalities in FMF.

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