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[Postural changes in systemic sclerosis: preliminary results].

P G Giacomini1, M Alessandrini, A Zoli

  • 1Clinica Otorinolaringoiatrica, Università di Roma Tor Vergata.

Acta Otorhinolaryngologica Italica : Organo Ufficiale Della Societa Italiana Di Otorinolaringologia E Chirurgia Cervico-Facciale
|January 5, 2002
PubMed
Summary
This summary is machine-generated.

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Systemic Sclerosis (SSc) patients exhibit subclinical postural control deficits, even without neurological symptoms. Posturography reveals widespread destabilization, indicating compromised postural mechanisms in SSc.

Area of Science:

  • Rheumatology and Neurology
  • Immunology
  • Biomechanical Engineering

Context:

  • Systemic Sclerosis (SSc) is a connective tissue disease often involving neurological complications.
  • Neurological involvement in SSc can manifest as neuropathies or unexplained postural alterations.
  • Previous studies have not comprehensively investigated postural control in SSc patients.

Purpose:

  • To evaluate static postural control in Systemic Sclerosis patients without subjective otoneurological symptoms.
  • To correlate posturographic findings with immunological markers and capillaroscopy in SSc.
  • To identify potential subclinical postural deficits in SSc.

Summary:

  • This study assessed 20 SSc patients and 10 controls using clinical examination, immunological tests, capillaroscopy, and computerized Static Posturography.

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  • Immunological tests revealed ANA positivity in all SSc patients, with specific antibodies detected.
  • Posturography showed significant increases in "surface" and "standard deviation of velocity" with eyes closed, and increased panfrequency oscillations in SSc patients.
  • Spectral frequency analysis indicated compromised postural control mechanisms, leading to destabilization with and without visual input.

    • Impact:

    • This research is believed to be the first to demonstrate subclinical postural control impairment in Systemic Sclerosis.
    • Findings suggest that SSc affects postural control mechanisms, contributing to destabilization.
    • Highlights the need for further investigation into the neurological underpinnings of postural instability in SSc.