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The heart rate response to exercise and circulating catecholamines in heart transplant recipients

Guido Ferretti1, Claudio Marconi, Guido Achilli

  • 1Département de Physiologie, Centre Médical Universitaire, 1 rue Michel Servet, 1211 Genève 4, Switzerland.

Insights

Heart transplant recipients (HTR) show a heart rate response to plasma catecholamines similar to healthy subjects, suggesting potential sympathetic reinnervation and beta-receptor modulation over time.

Area of Science:

  • Cardiovascular Physiology
  • Transplantation Medicine
  • Autonomic Nervous System

Background:

  • Plasma concentrations of noradrenaline ([NA]) and adrenaline ([A]) are higher in normal subjects and heart transplant recipients (HTR).
  • The denervated heart's chronotropic response to plasma [NA] and [A] is hypothesized to mimic beta1-adrenergic receptor stimulation due to higher myocardial beta1 than beta2 receptor density.

Purpose of the Study:

  • To investigate the chronotropic response of HTRs' hearts to changes in plasma [NA] and [A] during exercise.
  • To test the hypothesis that the denervated heart's response mimics beta1-adrenergic receptor stimulation.

Main Methods:

  • 17 HTRs and 9 healthy controls (CTL) underwent incremental exercise on a cycle ergometer.
  • Heart rate (HR) was monitored via electrocardiography.
  • Plasma [NA] and [A] were measured using high-pressure liquid chromatography at rest and varying workloads.

Main Results:

  • In both groups, HR and plasma catecholamine levels ([NA+A]) increased with workload.
  • Normalized HR values correlated significantly with the logarithm of [NA+A] in both HTRs and CTLs.
  • Affinity constants for catecholamine response differed between HTRs (2599+/-350 ng.l(-1)) and CTLs (487+/-37 ng.l(-1)).

Conclusions:

  • The chronotropic effect of plasma catecholamines in HTRs resembles combined beta1 and beta2 adrenergic activation.
  • Findings suggest potential sympathetic reinnervation and beta-receptor modulation in HTRs over time.
  • The hypothesis of persistent heart denervation in HTRs is challenged.

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