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[Hyperlipoproteinemia and LDL apheresis. Clinical experiences with the H.E.L.P. system]

B R Jaeger1, D Seidel

  • 1Institut für Klinische Chemie am Klinikum Grosshadern, Ludwig-Maximilians-Universität München.

Herz
|February 1, 2002
PubMed

Insights

LDL-cholesterol (LDL) is a key factor in atherosclerosis and coronary heart disease (CHD). Apheresis therapies, like the H.E.L.P.-system, effectively reduce LDL and other risk factors in patients resistant to standard treatments.

Area of Science:

  • Cardiovascular Medicine
  • Lipidology
  • Medical Technology

Context:

  • High levels of low-density lipoprotein cholesterol (LDL-C) are a primary cause of atherosclerosis and coronary heart disease (CHD).
  • Therapeutic guidelines aim for LDL-C levels below 100 mg/dl.
  • Statins effectively lower LDL-C in most patients, reducing CHD incidence and mortality.

Purpose:

  • To review the role of apheresis in managing patients with hyperlipidemia resistant to conventional therapies.
  • To highlight the efficacy and safety of the H.E.L.P.-apheresis system.

Summary:

  • LDL-C elevation is a major risk factor for atherosclerosis and CHD.
  • While statins are effective, some patients exhibit resistance to treatment.
  • Apheresis, particularly the H.E.L.P.-system, offers an effective alternative for LDL-C reduction, also removing Lp(a), Fibrinogen, and CRP.
  • Clinical data show significant risk factor reduction and clinical event decrease with long-term apheresis tolerance.

Impact:

  • Apheresis provides a viable therapeutic option for refractory hyperlipidemia, reducing cardiovascular risk.
  • The H.E.L.P.-system demonstrates broad efficacy in removing multiple atherogenic and inflammatory markers.
  • Long-term apheresis use is well-tolerated, contributing to improved patient outcomes.

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