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Parameters of inflammation and infection in a community based case-control study of coronary heart disease

J De Backer1, R Mak, D De Bacquer

  • 1Department of Cardiology, University Hospital, Ghent, Belgium.

Atherosclerosis
|February 19, 2002
PubMed

Insights

Coronary heart disease (CHD) is linked to higher inflammation markers like C-reactive protein (CRP), independent of other risk factors. No link was found between these inflammation markers and common infectious agents in men with CHD.

Area of Science:

  • Cardiology
  • Immunology
  • Epidemiology

Background:

  • Systemic inflammatory markers are associated with coronary heart disease (CHD).
  • The role of confounding factors, such as coronary risk factors and chronic infections, in this association is unclear.

Purpose of the Study:

  • To investigate differences in inflammatory markers (CRP, SAA, fibrinogen) and infectious agent serostatus between men with stable CHD and controls, independent of traditional risk factors.
  • To examine the relationship between inflammatory markers and serostatus for four infectious agents (H. pylori, C. pneumoniae, CMV, EBV) in CHD patients and healthy controls.

Main Methods:

  • A large-scale epidemiologic survey compared 446 men with CHD history to 892 matched controls.
  • Measurements included inflammatory biomarkers (CRP, fibrinogen, SAA) and antibodies against H. pylori, C. pneumoniae, CMV, and EBV, alongside classical coronary risk factors.

Main Results:

  • Univariate analysis showed higher CRP, fibrinogen, and SAA in CHD cases, but no differences in infectious agent serostatus.
  • Multivariate analysis confirmed significantly higher CRP in CHD cases, independent of other risk factors and medications.
  • No association was found between inflammatory biomarkers and serostatus for any of the four infectious agents.

Conclusions:

  • Elevated C-reactive protein (CRP) levels are independently associated with coronary heart disease (CHD) in working men.
  • No link exists between inflammatory biomarkers and serostatus for H. pylori, C. pneumoniae, CMV, or EBV in this population.
Abstract

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