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Cardiopulmonary exercise parameters in children with atrial septal defect and increased pulmonary blood flow:

J P Pfammatter1, M Zanolari, A Schibler

  • 1Pediatric Cardiology, University Children's Hospital, Berne, Switzerland. jean.pierre.pfammatter@insel.ch

Insights

Children with atrial septal defects (ASD) show impaired exercise capacity and increased airway resistance at peak exercise. Shunt closure did not fully restore maximum oxygen uptake or chronotropic response, indicating persistent cardiorespiratory effects.

Area of Science:

  • Pediatric Cardiology
  • Cardiopulmonary Physiology

Background:

  • Atrial septal defects (ASD) cause increased pulmonary blood flow, potentially impairing cardiopulmonary function during exercise.
  • Limited data exist on the short-term effects of ASD closure on cardiorespiratory function at peak exercise in children.

Purpose of the Study:

  • To prospectively evaluate preoperative and postoperative cardiorespiratory function at peak exercise in children with ASD.
  • To compare these parameters with a matched healthy population.

Main Methods:

  • Prospective study involving pulmonary function testing and cardiopulmonary exercise testing.
  • Hemodynamic assessment preoperatively and 3-4 months postoperatively in 16 children with ASD.
  • Comparison with 15 age-matched healthy children.

Main Results:

  • Preoperatively, ASD patients showed no significant differences in baseline pulmonary function or exercise capacity compared to controls.
  • At peak exercise, ASD patients exhibited increased pulmonary resistance, particularly in distal airways, and elevated serum lactate.
  • Maximum oxygen uptake and chronotropic response to exercise were impaired in ASD patients, persisting postoperatively.

Conclusions:

  • Cardiopulmonary exercise parameters in children with ASD showed only slight deviations from normal children.
  • Key differences included reduced maximum oxygen uptake and increased airway resistance during maximal exercise.
  • These findings highlight persistent cardiorespiratory limitations even after shunt closure.
Abstract

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