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Comparative pathogenicity of three genetically distinct Trypanosoma congolense-types in inbred Balb/c mice

Z Bengaly1, I Sidibe, H Boly

  • 1CIRDES-Centre International de Recherche-Developpement sur l'Elevage en Zone subhumide, 01BP 454, Bobo-Dioulasso, Burkina Faso. toure@ouaga.ird.bf

Insights

African trypanosomosis pathogenicity varies by Trypanosoma congolense type. Savannah types are highly virulent, causing rapid death in mice, while forest and kilifi types exhibit low virulence and chronic infections.

Area of Science:

  • Veterinary Parasitology
  • Molecular Epidemiology
  • Disease Pathogenesis

Background:

  • African trypanosomosis is a significant disease affecting livestock and humans.
  • Trypanosoma congolense exists in genetically distinct types, influencing disease presentation.
  • Understanding type-specific pathogenicity is crucial for epidemiological control.

Purpose of the Study:

  • To compare the pathogenicity of different Trypanosoma congolense types in mice.
  • To investigate potential strain variations within the savannah type.
  • To enhance the understanding of African trypanosomosis epidemiology.

Main Methods:

  • Infection of inbred Balb/c mice with distinct Trypanosoma congolense clones (savannah, forest, kilifi).
  • Inclusion of an additional savannah clone (IL 3000) to assess strain variation.
  • Monitoring of clinical signs, parasitaemia, prepatent period, and mortality rates.

Main Results:

  • Savannah type clones (Sam.28.1, IL 3000) demonstrated extreme virulence, leading to rapid mortality within a week.
  • Forest (Dind.3.1) and kilifi (K60.1A) type clones exhibited low virulence, resulting in chronic infections and minimal mortality.
  • Rapidly evolving parasitaemia in virulent infections correlated positively with the prepatent period.

Conclusions:

  • Trypanosoma congolense type significantly influences pathogenicity and disease course in mice.
  • Savannah type strains are highly virulent, posing a greater immediate threat.
  • Forest and kilifi types cause chronic, less severe infections, impacting long-term disease dynamics.

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