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Glucose-6-phosphate dehydrogenase deficiency and hematopoietic stem cell transplantation

W Y Au1, S K Ma, A K W Lie

  • 1Department of Medicine, University of Hong Kong, Queen Mary Hospital, Hong Kong.

Insights

Glucose-6-phosphate dehydrogenase (G6PD) deficiency is an X-linked condition. Testing all stem cell transplant donors and recipients for G6PD deficiency is recommended, especially in susceptible populations, due to its potential impact.

Area of Science:

  • Hematology
  • Genetics
  • Enzymology

Background:

  • Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a common X-linked enzymopathy.
  • It affects approximately 3% of Southern Chinese males, presenting as a hemolytic condition.

Purpose of the Study:

  • To investigate the incidence and clinical implications of G6PD deficiency in adult hematopoietic stem cell transplantation (SCT) recipients and donors.
  • To assess the impact of G6PD deficiency on engraftment and hemolysis post-SCT.
  • To explore the prevalence of G6PD deficiency in female patients with chronic myeloid leukemia (CML).

Main Methods:

  • Retrospective analysis of 275 allogeneic and 107 autologous SCT cases.
  • Screening for G6PD deficiency in both donors and recipients.
  • Monitoring for hemolysis and red cell engraftment post-transplantation.
  • Statistical comparison of G6PD deficiency incidence in different patient groups.

Main Results:

  • G6PD deficiency was identified in 1.8% of allogeneic SCT pairs and 3.7% of autologous SCT patients.
  • Female CML patients showed a significantly higher incidence of G6PD deficiency compared to the general female population.
  • No significant hemolysis or delayed engraftment was observed in most G6PD-deficient individuals; most converted to donor G6PD status post-SCT.

Conclusions:

  • G6PD deficiency screening for all SCT donors and recipients in susceptible populations is advisable.
  • There is a potential association between G6PD deficiency and multi-lineage clonal marrow disorders, particularly in females.
  • While generally well-tolerated post-SCT, G6PD deficiency warrants consideration in transplant protocols.

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