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Behaviour, chemosignals and endocrine functions in male mice infected with tick-borne encephalitis virus

Mikhail Moshkin1, Ludmila Gerlinskaya, Olga Morozova

  • 1Institute of Systematics and Ecology of Animals, Siberian Branch of RAS, 630091, Novosibirsk, Frunze str. 11, Russia. mmp@eco.nsc.ru

Insights

Tick-borne encephalitis virus (TBE) infection in male mice increases testosterone, enhancing their attractiveness to females and aggression. This benefits TBE virus transmission within the host population.

Area of Science:

  • Virology
  • Neuroendocrinology
  • Animal Behavior

Background:

  • Subclinical infections can alter host physiology and behavior.
  • Understanding how viruses manipulate host behavior is crucial for disease ecology.

Purpose of the Study:

  • To investigate the effects of tick-borne encephalitis virus (TBE) infection on male mouse endocrine status, social behavior, and olfactory attractiveness.

Main Methods:

  • Male ICR mice were inoculated with TBE virus.
  • Mice were categorized into TBE-positive (viral RNA detected) and TBE-negative subgroups using RT-PCR.
  • Plasma testosterone levels, odor attractiveness to receptive females, and aggressiveness in social conflicts were assessed.

Main Results:

  • TBE-positive males exhibited significantly higher plasma testosterone levels compared to controls and TBE-negative males.
  • TBE-positive males displayed increased olfactory attractiveness to females and heightened aggressiveness during social interactions.
  • These behavioral changes were observed 6-7 days post-infection.

Conclusions:

  • Subclinical TBE virus infection can enhance male mouse testosterone levels, leading to increased sexual attractiveness and aggression.
  • These virus-induced behavioral modifications may facilitate pathogen transmission by increasing host contact rates.

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