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Creatine phosphokinase following cardioversion.

G Forssell, R Nordlander, O Nyquist

    Acta Medica Scandinavica
    |November 1, 1975
    PubMed
    Summary
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    Cardioversion for supraventricular tachyarrhythmias rarely impacts diagnosing acute myocardial infarction (AMI) using creatine phosphokinase (CPK) levels. CPK levels in patients without AMI remained within normal limits post-cardioversion.

    Area of Science:

    • Cardiology
    • Biochemistry

    Background:

    • Supraventricular tachyarrhythmias require treatment like cardioversion.
    • Acute myocardial infarction (AMI) diagnosis relies on serial creatine phosphokinase (CPK) measurements.
    • The potential impact of cardioversion on CPK levels is a diagnostic concern.

    Purpose of the Study:

    • To evaluate the effect of cardioversion on serum CPK levels.
    • To determine if cardioversion interferes with diagnosing AMI using CPK.
    • To assess CPK changes in patients undergoing cardioversion for supraventricular tachyarrhythmias.

    Main Methods:

    • Serial total serum CPK estimations were performed before and up to 51 hours after cardioversion.
    • 12 patients without AMI undergoing cardioversion were studied.

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  • CPK levels were compared to normal limits and levels observed in AMI patients.

    • Main Results:

    • The maximal CPK rise observed was 78 mU/ml (110%), not exceeding normal limits in most patients.
    • Only two patients had CPK levels slightly above the normal limit (149 and 156 mU/ml).
    • This contrasts with significantly higher CPK elevations (mean 900 mU/ml) observed in AMI patients.

    Conclusions:

    • Cardioversion for supraventricular tachyarrhythmias generally does not interfere with diagnosing AMI via serial CPK estimations within 24 hours.
    • The observed CPK elevations post-cardioversion are typically minor and distinct from those seen in AMI.
    • Serial CPK monitoring remains a reliable tool for AMI diagnosis even after cardioversion.