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VCAM-1-positive microglia target oligodendrocytes at the border of multiple sclerosis lesions

John W Peterson1, Lars Bö, Sverre Mörk

  • 1Department of Neurosciences, Lerner Research Institute, The Cleveland Clinic Foundation, Ohio 44195, USA.

Insights

Vascular cell adhesion molecule-1 (VCAM-1) positive macrophages/microglia cluster at active multiple sclerosis (MS) lesion borders, interacting with myelin-producing cells. This study reveals direct microglial contact with oligodendrocytes during MS demyelination.

Area of Science:

  • Neuroimmunology
  • Cellular Biology
  • Neuropathology

Background:

  • Multiple sclerosis (MS) is a chronic inflammatory demyelinating disease of the central nervous system.
  • The role of specific cell adhesion molecules and immune cell interactions in MS lesion pathogenesis remains incompletely understood.

Purpose of the Study:

  • To investigate the distribution and lineage of vascular cell adhesion molecule-1 (VCAM-1)-positive cells in multiple sclerosis (MS) brain lesions.
  • To determine the spatial relationship between VCAM-1-positive cells and oligodendrocytes in active MS lesions.

Main Methods:

  • Immunohistochemical analysis of VCAM-1 expression in brain tissue from 43 MS patients.
  • Quantitative assessment of VCAM-1-positive cells in active, chronic active, and chronic inactive MS lesions.
  • Microscopic examination of cell-cell interactions at lesion borders.

Main Results:

  • Numerous VCAM-1-positive macrophages/microglia were found at the actively demyelinating borders of active and chronic active MS lesions.
  • VCAM-1-positive cells were rarely detected in chronic inactive lesions.
  • Approximately 17% of VCAM-1-positive cells were closely associated with or surrounding oligodendrocyte perikarya in active lesions.

Conclusions:

  • VCAM-1 expression is prominent on macrophages/microglia at active MS lesion sites.
  • These VCAM-1-positive myeloid cells engage directly with oligodendrocytes in areas of active demyelination.
  • This study provides the first evidence of direct microglial interaction with oligodendrocytes in the context of multiple sclerosis.

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