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[Functional dyspepsia--a psychosomatic disease].

Tone Tangen Haug1

  • 1Psykiatrisk institutt Haukeland Sykehus 5021 Bergen. mphth@pop3.uib.no

Tidsskrift for Den Norske Laegeforening : Tidsskrift for Praktisk Medicin, Ny Raekke
|July 2, 2002
PubMed
Summary

Functional dyspepsia, often diagnosed when no ulcer is found, is primarily caused by gastric motility issues and hypersensitivity, not H. pylori. Antidepressants and psychological therapies effectively reduce symptoms, unlike traditional treatments.

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Journal of psychosomatic research·2002

Area of Science:

  • Gastroenterology
  • Psychiatry
  • Internal Medicine

Context:

  • Upper gastrointestinal complaints represent 4% of general practice consultations.
  • Peptic ulcer disease is identified in only 20-30% of patients with these symptoms.
  • Endoscopy reveals no organic cause in 20-50% of cases, leading to a diagnosis of functional dyspepsia.

Purpose:

  • To elucidate the primary pathophysiological mechanisms of functional dyspepsia.
  • To evaluate the efficacy of various treatment modalities for functional dyspepsia.

Summary:

  • Functional dyspepsia is characterized by gastric motor abnormalities and visceral hypersensitivity, rather than hypersecretion of gastric acid or Helicobacter pylori infection.
  • Stress, anxiety, and depression are strongly linked to the development and exacerbation of functional dyspepsia.

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  • Pharmacological treatments like antacids, H2-blockers, and prokinetics show limited efficacy, often comparable to placebo.

    • Impact:

    • Antidepressants and psychological interventions (e.g., cognitive behavioral therapy, stress management) are effective in alleviating dyspeptic symptoms.
    • This research highlights the importance of addressing psychological factors and motility disorders in managing functional dyspepsia.
    • Findings suggest a shift in treatment strategies for functional dyspepsia, emphasizing psychotherapeutic and psychotropic approaches over traditional gastrointestinal medications.