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Echogenic bowel in intrauterine growth restriction fetuses: does this jeopardize the gut?

Reuwen Achiron1, Rami Mazkereth, Raoul Orvieto

  • 1Department of Obstetrics and Gynecology, The Chaim Sheba Medical Center, Tel Hashomer, Israel. rachiron@post.tau.ac.il

Insights

Fetal echogenic bowel in fetuses with intrauterine growth restriction (IUGR) does not increase the risk of necrotizing enterocolitis. These IUGR fetuses show vasodilatation in the superior mesenteric artery and celiac trunk.

Area of Science:

  • Perinatal Medicine
  • Neonatal Surgery
  • Fetal Medicine

Background:

  • Intrauterine growth restriction (IUGR) is a significant concern in high-risk pregnancies.
  • Echogenic bowel and umbilical artery high resistance are potential indicators of fetal compromise.
  • Neonatal necrotizing enterocolitis (NEC) is a severe gastrointestinal emergency in newborns.

Purpose of the Study:

  • To determine if IUGR fetuses with echogenic bowel and high umbilical artery resistance are at increased risk for NEC.
  • To investigate fetal vascular changes in IUGR fetuses with specific sonographic findings.

Main Methods:

  • Comparison of two groups: singleton IUGR fetuses (echogenic bowel or reversed diastolic flow) and neonates diagnosed with NEC.
  • Doppler velocimetry of the superior mesenteric artery and celiac trunk in IUGR fetuses.
  • Retrospective analysis of medical records for neonates with NEC.

Main Results:

  • None of the IUGR fetuses with echogenic bowel developed NEC.
  • The mean birth weight was significantly lower in the IUGR group compared to the NEC group.
  • IUGR fetuses demonstrated significantly lower pulsatility indices in the superior mesenteric artery and celiac trunk, indicating vasodilatation.

Conclusions:

  • Fetal echogenic bowel in IUGR fetuses is not associated with an increased risk of developing NEC.
  • IUGR fetuses with echogenic bowel exhibit vasodilatation in the superior mesenteric artery and celiac trunk.
  • These findings suggest a different pathophysiological pathway than previously assumed for NEC development in IUGR.
Abstract

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