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Endothelium-specific platelet-derived growth factor-B ablation mimics diabetic retinopathy

Maria Enge1, Mattias Bjarnegård, Holger Gerhardt

  • 1Department of Medical Biochemistry, Göteborg University, PO Box 440, SE 405 30 Göteborg, Sweden.

The EMBO Journal
|August 10, 2002
PubMed

Insights

Pericyte loss from retinal capillaries is linked to diabetic retinopathy. This study shows reduced pericyte density in mice causes retinopathy, suggesting pericyte loss is a key factor in human diabetic eye disease.

Area of Science:

  • Ophthalmology
  • Vascular Biology
  • Diabetic Complications

Background:

  • Loss of pericytes from retinal capillaries is a hallmark of diabetic retinopathy.
  • The pathogenic role of pericyte loss in diabetic retinopathy remains unclear.
  • Previous studies were limited by prenatal lethality in mouse models of pericyte deficiency.

Purpose of the Study:

  • To investigate the consequences of pericyte deficiency on retinal microvasculature.
  • To determine if reduced pericyte density is sufficient to cause retinopathy.
  • To establish a viable mouse model for studying pericyte loss in the retina.

Main Methods:

  • Endothelium-restricted ablation of platelet-derived growth factor-B (PDGF-B) in mice.
  • Analysis of pericyte density and retinal microvascular abnormalities.
  • Correlation of pericyte density with retinopathy development.

Main Results:

  • Generated viable mice with variable pericyte densities in the central nervous system (CNS).
  • Found a strong inverse correlation between pericyte density and retinal microvascular abnormalities.
  • Pro-liferative retinopathy developed when pericyte density fell below 50% of normal.

Conclusions:

  • Reduced pericyte density is sufficient to induce retinopathy in mice.
  • Pericyte loss may be a causal pathogenic event in human diabetic retinopathy.
  • This model allows for further investigation into the mechanisms of diabetic retinopathy.

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