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Novel ceftazidime-resistance beta-lactamases generated by a codon-based mutagenesis method and selection

Paul Gaytán1, Joel Osuna, Xavier Soberón

  • 1Instituto de Biotecnología/UNAM, Ap. Postal 510-3, Cuernavaca, Morelos 62250, México. paul@ibt.unam.mx

Nucleic Acids Research
|August 15, 2002
PubMed

Insights

A novel codon-based mutagenesis method efficiently generated 13 beta-lactamases, including new ceftazidime-resistant variants. This approach enables rapid enzyme evolution for antibiotic resistance research.

Area of Science:

  • Molecular Biology
  • Enzyme Engineering
  • Drug Discovery

Background:

  • Beta-lactamase enzymes confer resistance to antibiotics like ceftazidime.
  • Developing novel beta-lactamases is crucial for understanding and combating antibiotic resistance.
  • Existing mutagenesis methods can be inefficient for generating diverse enzyme variants.

Purpose of the Study:

  • To develop and validate a novel, automated codon-based mutagenesis approach.
  • To generate a library of ceftazidime-resistance beta-lactamases.
  • To identify enhanced beta-lactamase variants with significant ceftazidime resistance.

Main Methods:

  • A contaminating codon-based mutagenesis strategy was employed during oligonucleotide synthesis.
  • Mutant and wild-type codons were combined in a single synthesis column for automation.
  • The method was tested using plasmid pUC19 and applied to TEM-1 beta-lactamase mutagenesis.

Main Results:

  • Four known and nine new beta-lactamase variants were successfully generated.
  • The mutagenesis approach demonstrated random and binomial behavior.
  • A triple mutant (R164H:E240K:R241A) showed a 333-fold increase in ceftazidime resistance compared to wild-type.

Conclusions:

  • The novel codon-based mutagenesis method is efficient and automatable for generating enzyme variants.
  • This approach accelerates the discovery of enzymes with enhanced antibiotic resistance properties.
  • The identified beta-lactamase variants contribute to understanding ceftazidime resistance mechanisms.

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