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Characterization of enolase allergen from Rhodotorula mucilaginosa

Ching-Yun Chang1, Hong Chou, Ming F Tam

  • 1Department of Medical Research and Education, Veterans General Hospital-Taipei, Taipei, Taiwan, ROC.

Insights

This study identifies a new enolase allergen, Rho m 1, from Rhodotorula mucilaginosa, a common yeast. It reveals cross-reactivity among fungal enolases, crucial for understanding mould allergies.

Area of Science:

  • Molecular Allergology
  • Mycology
  • Immunology

Background:

  • Rhodotorula mucilaginosa (R. rubra) is a ubiquitous yeast, but its allergens remain uncharacterized at the molecular level.
  • Understanding fungal allergens is critical for diagnosing and managing allergic diseases, particularly mould allergies.

Purpose of the Study:

  • To molecularly characterize the enolase allergen from R. mucilaginosa.
  • To investigate the allergenic and antigenic cross-reactivity of R. mucilaginosa enolase with other fungal enolases.

Main Methods:

  • Isolation of full-length cDNA encoding R. mucilaginosa enolase using reverse transcriptase-polymerase chain reaction and rapid amplification of cDNA ends.
  • Identification of natural enolase via two-dimensional gel electrophoresis and N-terminal amino acid sequencing.
  • Assessment of IgE binding in allergic patients' sera and cross-reactivity using rabbit antiserum and monoclonal antibodies (MoAbs).

Main Results:

  • The enolase from R. mucilaginosa (Rho m 1) is a 439-residue protein encoded by a 1497 bp cDNA, showing high sequence identity to enolases from other fungi.
  • A 47-kD component in R. mucilaginosa extracts reacted with IgE and anti-enolase antibodies, confirming it as the enolase allergen.
  • Sera from 21% of allergic patients showed IgE binding to R. mucilaginosa enolase; cross-reactivity was observed among enolases from R. mucilaginosa, Candida albicans, and Penicillium citrinum.

Conclusions:

  • A novel, cross-reactive enolase allergen, Rho m 1, from R. mucilaginosa has been identified.
  • While fungal enolases are conserved, individual patient IgE reactivity varies significantly across different fungal species.
  • These findings contribute to understanding the role of enolase in clinical mould allergy.

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