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Identification of Staphylococcus xylosus isolated from C57BL/6J-Nos2(tm1Lau) mice with dermatitis

Young Suk Won1, Hyo Jung Kwon, Goo Taeg Oh

  • 1Department of Microbiology, College of Veterinary Medicine and School of Agricultural Biotechnology, Seoul National University, Suwon, Korea.

Microbiology and Immunology
|November 20, 2002
PubMed

Insights

Coagulase-negative staphylococci (CNS) can cause infections, especially in immunocompromised individuals. This study identifies Staphylococcus xylosus as a potential opportunistic pathogen in mice with dermatitis.

Area of Science:

  • Microbiology
  • Immunology
  • Veterinary Pathology

Background:

  • Coagulase-negative staphylococci (CNS) are recognized as significant pathogens, frequently implicated in medical device-associated infections and infections in immunocompromised patients.
  • Dermatitis is a common condition affecting various animal models, potentially leading to secondary bacterial infections.

Purpose of the Study:

  • To identify the bacterial species responsible for dermatitis in NOS2 knockout mice.
  • To evaluate the pathogenic potential of isolated CNS strains in an immunocompromised host model.

Main Methods:

  • Isolation and bacterial culture from skin lesions of affected mice.
  • Histopathological examination of skin biopsies to characterize dermatitis.
  • Bacterial identification using API STAPH system and 16S-23S intergenic transcribed spacer-PCR.

Main Results:

  • Five CNS strains were isolated from five NOS2 knockout mice exhibiting dermatitis.
  • Histopathology revealed granulomatous dermatitis with epidermal ulceration, pustules, necrosis, and inflammatory cell infiltration.
  • Bacterial isolates were identified as Staphylococcus xylosus.

Conclusions:

  • Staphylococcus xylosus was identified as the causative agent of dermatitis in this cohort of immunocompromised mice.
  • These findings highlight the opportunistic pathogenic potential of Staphylococcus xylosus, particularly in immunocompromised hosts.
  • The study provides insights into bacterial coinfections in genetically modified mouse models with inflammatory conditions.

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