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[Adaptation of thyroid function to excess iodine].

Andre Aurengo1, Laurence Leenhardt, Helyett Aurengo

  • 1Service de médecine nucléaire, Groupe hospitalier Pitié-Salpêtrière, 83 bd de l'Hôpital 75651 Paris.

Presse Medicale (Paris, France : 1983)
|November 27, 2002
PubMed
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Excess iodine can disrupt thyroid hormone production, especially in newborns and adults with existing thyroid conditions. While the thyroid usually adapts, iodine-induced dysthyroidism is a significant concern, particularly with amiodarone use.

Area of Science:

  • Endocrinology
  • Thyroid Physiology
  • Iodine Metabolism

Background:

  • Thyroid hormone production is typically stable despite varying iodine intake.
  • Iodine metabolism involves adapted thyroid uptake influenced by age and physiological status.
  • Iodine deficiency is prevalent in certain regions, while excess can pose risks.

Purpose of the Study:

  • To elucidate the mechanisms of thyroid adaptation to iodine excess.
  • To investigate the pathological impacts of iodine excess on thyroid function.
  • To understand the prevalence and causes of iodine-induced dysthyroidism.

Main Methods:

  • Review of recent advancements in thyroid physiology.
  • Analysis of clinical data on iodine-induced hyperthyroidism and hypothyroidism.

Related Experiment Videos

  • Examination of epidemiological data on iodine prophylaxis outcomes.

    • Main Results:

    • Mechanisms for adapting to iodine excess are immature in newborns and sometimes deficient in adults.
    • Iodine-induced hyperthyroidism is frequently linked to amiodarone.
    • Iodine-related hypothyroidism is common in pre-existing thyroid diseases and in newborns, especially premature infants.

    Conclusions:

    • Iodine excess can lead to dysthyroidism due to immature or deficient adaptive mechanisms.
    • Amiodarone is a significant factor in iodine-induced hyperthyroidism.
    • Iodine-related thyroid dysfunction is a notable concern in vulnerable populations like newborns.