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TACE mRNA expression in peripheral mononudear cells precedes new lesions on MRI in multiple sclerosis

T Seifert1, B C Kieseier, S Ropele

  • 1Department of Neurology, Karl Franzens University, Auenbruggerplatz 22, Graz A-8036, Austria. thomas.seifert@kfunigraz.ac.at

Multiple Sclerosis (Houndmills, Basingstoke, England)
|December 12, 2002
PubMed

Insights

Tumor necrosis factor-alpha (TNF-alpha) is key in multiple sclerosis (MS) pathogenesis. TACE mRNA in patient blood cells correlated with new MRI lesions, indicating disease activity.

Area of Science:

  • Neuroimmunology
  • Molecular Biology
  • Enzymology

Background:

  • Tumor necrosis factor-alpha (TNF-alpha) plays a role in multiple sclerosis (MS) pathogenesis.
  • TNF-alpha is released from its precursor by TNF-alpha-converting enzyme (TACE, ADAM 17).

Purpose of the Study:

  • To investigate the relationship between TACE mRNA expression in peripheral blood mononuclear cells (PBMCs) and MS disease activity.
  • To qualitatively assess mRNA levels of TNF-alpha and TACE in relapsing-remitting MS patients.

Main Methods:

  • Longitudinal study involving 11 relapsing-remitting MS patients.
  • Qualitative mRNA expression analysis of TNF-alpha and TACE in PBMCs without ex vivo stimulation.
  • Correlation of mRNA expression with monthly gadolinium (Gd)-enhanced brain MRI scans.

Main Results:

  • Patients positive for TACE mRNA in PBMCs showed a significantly higher mean number of new Gd-enhancing lesions per scan.
  • One month following PBMC sampling, TACE mRNA positivity was linked to increased MRI-detected disease activity.

Conclusions:

  • TACE mRNA expression in PBMCs may serve as a biomarker for predicting MS disease activity.
  • This finding highlights the potential role of TACE in MS pathogenesis and disease monitoring.

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