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Catatonia and ECT: Meduna's biological antagonism hypothesis reconsidered
1Department of Psychiatry, School of Medicine, SUNY at Stony Brook, Albert Einstein College of Medicine, P.O. Box 457, St. James, New York 11780-0457, USA. mafink@attglobal.net
Abstract:
Catatonia is a motor syndrome with abnormalities in neuroendocrine regulation. Both the motor syndrome and the neuroendocrine abnormalities respond rapidly to electroconvulsive therapy (ECT). This rapid efficacy supports a neuroendocrine hypothesis of the mode of action of ECT and encourages neuroendocrine studies of catatonia.