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Identification of UNC119 as a novel activator of SRC-type tyrosine kinases

Osman Cen1, Magdalena M Gorska, Susan J Stafford

  • 1Division of Allergy and Immunology, NIAID, National Institutes of Health Asthma and Allergic Diseases Research Center, Bethesda, Maryland 20892, USA.

Insights

A novel protein, Unc119, activates Lyn tyrosine kinase, crucial for eosinophil survival. This discovery reveals a new mechanism for regulating Src family kinases in immune cells.

Area of Science:

  • Immunology
  • Cell Signaling
  • Biochemistry

Background:

  • Lyn, an Src-type tyrosine kinase, is linked to the interleukin-5 (IL-5) receptor in eosinophils, but its activation mechanism remains unclear.
  • Src family tyrosine kinases play critical roles in cellular processes, including immune cell function and survival.

Purpose of the Study:

  • To identify and characterize novel signaling molecules involved in Lyn kinase activation within eosinophils.
  • To elucidate the mechanism by which Unc119 interacts with and activates Src family kinases, specifically Lyn.

Main Methods:

  • Yeast two-hybrid screening was employed to identify interacting partners of the IL-5 receptor alpha subunit and Src family kinases.
  • Biochemical assays were used to assess the catalytic activity of Lyn kinase upon interaction with Unc119.
  • Eosinophil transduction with Unc119 and subsequent survival assays were performed to evaluate its functional role.

Main Results:

  • A new signaling molecule, Unc119, was identified and characterized for its association with IL-5Ralpha and Src family kinases.
  • Unc119 was shown to induce catalytic activity in Lyn kinase through interactions with its Src homology 2 and 3 domains.
  • IL-5 stimulation enhanced Unc119-Lyn association and Lyn activity, leading to prolonged eosinophil survival, even without IL-5.

Conclusions:

  • Unc119 is identified as the first known receptor-associated activator of Src family tyrosine kinases.
  • Unc119 plays a significant role in eosinophil survival by modulating Lyn kinase activity.
  • This finding provides a new mechanistic insight into the regulation of immune cell signaling and survival pathways.

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