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Middle interhemispheric variant of holoprosencephaly: a distinct cliniconeuroradiologic subtype

A J Lewis1, E M Simon, A J Barkovich

  • 1Stanford University School of Medicine and Lucile Packard Children's Hospital, CA, USA.

Neurology
|December 25, 2002
PubMed
Abstract

Insights

Middle interhemispheric variant (MIH) holoprosencephaly differs from classic forms by lacking endocrine dysfunction and choreoathetosis. MIH patients show similar functional outcomes to the lobar subtype of holoprosencephaly.

Area of Science:

  • Neurology
  • Developmental Biology
  • Genetics

Background:

  • Middle interhemispheric variant (MIH) is a subtype of holoprosencephaly (HPE) characterized by incomplete midline separation in posterior brain regions.
  • While neuroradiologic features of MIH are known, its clinical manifestations remain largely uncharacterized.

Purpose of the Study:

  • To delineate the clinical features of MIH.
  • To compare the clinical presentation of MIH with classic HPE subtypes (alobar, semilobar, lobar).

Main Methods:

  • A multicenter study involving 15 patients with MIH.
  • Correlation of neuroimaging and clinical data.
  • Comparison with clinical and imaging data from 68 patients with classic HPE.

Main Results:

  • MIH patients exhibited a significantly lower frequency of endocrinopathy (0%) compared to classic HPE subtypes (72%), correlating with absent hypothalamic abnormalities.
  • Seizure incidence (40%) and spasticity (86%) in MIH were comparable to classic HPE.
  • Choreoathetosis was absent in MIH, contrasting with semilobar HPE (41%), linked to normal basal ganglia development.
  • Functional outcomes in MIH, including mobility, upper-extremity function, and language, were similar to lobar HPE.

Conclusions:

  • MIH represents a distinct subtype of HPE with a unique clinical profile and prognosis.
  • MIH is differentiated from classic HPE by the absence of endocrine dysfunction and choreoathetosis.
  • Functional outcomes align MIH with the milder lobar HPE subtype.

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