[Post-thyroidectomy hemorrhage]

Stanisław Cichoń1, Ryszard Anielski, Paweł Orlicki

  • 1Klinika Chirurgii Endokrynologicznej III Katedry Chirurgii Collegium Medicum Uniwersytetu Jagiellońskiego w Krakowie.

Przeglad Lekarski
|January 9, 2003
PubMed

The aim of the study was to estimate the prevalence and causes of haemorrhages, that required extemporaneous surgical treatment, after goiter operations, in our own material. Between 1984 and 2000, i.e. during 17 years, 7400 patients were operated in our Clinic due to different kinds of goiter. Haemorrhage during post operative period was presented in 46 (0.62%) patients. There were 37 women and 9 men in the analysed material. The age of patients was between 19 and 77 years, and the average age was 50.6 +/- 15.4. The lowest percentage of this complication was observed after operation of non-toxic goiter (0.39%) and the highest in a group of patients operated due to Graves-Basedow's disease (1.02%). Frequent local symptoms were observed, such as: enlargement of circumference of neck, supple haematoma with dyspnoea, and sometimes swallowing difficulties. A decrease in blood pressure was observed in 5 (10.9%) patients. Low molecular weight heparin prophylaxis was applied in 18 (39.1%) patients and in 2 cases disorders of blood coagulability were observed. The quantity of drainage from the wound was between 30 and 610 ml, with average 249.8 +/- 144.69 ml. Time from main operation termination to revision was 20 to 1440 minutes, with average time 346 +/- 285.66 minutes. The most common cause of post-thyroidectomy haemorrhage was bleeding from the thyroid lobe stump, which presented in 28 (60.9%) patients. The most dangerous haemorrhage, because of its intensity, was the bleeding from the upper thyroid artery (6 patients). During the postoperative period 16 patients (34.8%) required blood transfusions, however massive transfusions (4 and more blood units) were performed only in 4 patients. Early complications included: paralysis of recurrent laryngeal nerve in 6 patients and transient hypocalcaemia in one patient.

Related Concept Videos

Hyperthyroidism II: Pathophysiology01:27

Hyperthyroidism II: Pathophysiology

Hyperthyroidism is a hypermetabolic state caused by elevated levels of thyroid hormones, triiodothyronine (T3) and thyroxine (T4). It results from dysregulation at the thyroid, pituitary, or immune system level and affects multiple organ systems.PathophysiologyThe most common cause of hyperthyroidism is Graves’ disease, an autoimmune disorder in which antibodies, specifically thyroid-stimulating antibodies (TSAb), a subtype of TSH receptor antibodies (TRAb), bind to and activate TSH receptors...
Graves Disease II: Pathophysiology01:24

Graves Disease II: Pathophysiology

Graves’ disease is an autoimmune disorder characterized by the production of thyroid-stimulating immunoglobulins (TSI) that activate TSH receptors, leading to excessive synthesis and release of thyroid hormones (T3 and T4) and resulting in hyperthyroidism.Among all causes of hyperthyroidism, Graves’ disease is the most common and can happen at any age, though it is more frequent in women. It produces a hypermetabolic state with features such as weight loss, tachycardia, tremor, and heat...
Aneurysm IV: Nursing Management01:22

Aneurysm IV: Nursing Management

Vigilant monitoring for aneurysm rupture is essential for patients undergoing aortic surgery.Preoperative Nursing ManagementContinuously monitor the patient for manifestations of aneurysm rupture, such as pallor, weakness, tachycardia, hypotension, abdominal, back, groin, or periumbilical pain, changes in consciousness, and a pulsating abdominal mass. Regularly assess the patient's peripheral pulses.Instruct the patient to consume a clear liquid diet the day before surgery and administer...
Graves' Disease I: Introduction01:28

Graves' Disease I: Introduction

Graves' disease is an autoimmune disorder that causes hyperthyroidism, or overactivity of the thyroid gland. It results from autoantibodies called thyroid-stimulating immunoglobulins (TSIs), which bind to thyroid-stimulating hormone (TSH) receptors, leading to overstimulation of hormone production and a hypermetabolic state.EtiologyAlthough considered idiopathic, Graves’ disease has well-established contributing factors. There is a strong genetic component, with increased prevalence in...
Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Esophageal Varices-II: Clinical Features and Management01:28

Esophageal Varices-II: Clinical Features and Management

Esophageal varices often manifest as gastrointestinal bleeding episodes, presenting symptoms like hematemesis (vomiting of blood), hematochezia (passing fresh blood via the rectum), and melena (black, tarry stools). Other signs can include weight loss, anorexia, abdominal discomfort, jaundice, pruritus, altered mental status, and muscle cramps.
In the initial assessment, a thorough review of the patient's medical history is vital to identify risk factors such as liver disease, alcohol abuse, or...