Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Delayed encephalopathy after strangling.

E C Dooling, E P Richardson

    Archives of Neurology
    |March 11, 1976
    PubMed
    Summary
    This summary is machine-generated.

    A child developed severe neurological symptoms including involuntary movements and opisthotonos one week after strangulation. Neuropathology revealed bilateral brain lesions, suggesting a potential metabolic disorder like lactic acidosis.

    Related Concept Videos

    You might also read

    Related Articles

    Articles linked to this work by shared authors, journal, and citation graph.

    Sort by
    Same author

    The Vascular Access Questionnaire: assessing patient-reported views of vascular access.

    The journal of vascular access·2008
    Same author

    Home care assistance and the utilization of peritoneal dialysis.

    Kidney international·2007
    Same author

    Diffusion-weighted imaging in moyamoya disease.

    Journal of child neurology·2001
    Same author

    The neurologic illness of Eugene O'Neill--a clinicopathological report.

    The New England journal of medicine·2000
    Same author

    Multifocal embolic strokes following cardiac surgery in a protein C-deficient child.

    Journal of child neurology·1999
    Same author

    Moyamoya and Down syndrome. Clinical and radiological features.

    Stroke·1996
    Same journal

    Incorrect Table Entries and Word.

    Archives of neurology·2016
    Same journal

    IDEAL for CCSVI Research-Reply.

    Archives of neurology·2013
    Same journal

    Atlas of Inherited Metabolic Diseases, 3rd ed.

    Archives of neurology·2013
    Same journal

    Error in byline: in heterogeneity of coenzyme q10 deficiency: patient study and literature review.

    Archives of neurology·2013
    Same journal

    This month in archives of neurology.

    Archives of neurology·2013
    Same journal

    About this journal.

    Archives of neurology·2013
    See all related articles

    Area of Science:

    • Neurology
    • Pathology
    • Toxicology

    Background:

    • Hypoxic-ischemic insult can lead to delayed-onset neurological disorders.
    • Strangulation is a form of hypoxic-ischemic insult with potential for delayed neurological sequelae.
    • Previous cases demonstrate varied clinical presentations following strangulation.

    Observation:

    • An 11-year-old boy presented with involuntary movements, opisthotonos, and autonomic dysfunction one week post-strangulation.
    • The patient remained alert with normal electroencephalogram (EEG) initially.
    • Neuropathological examination revealed bilateral cavitating lesions in the basal ganglia (caudate nucleus, putamen, globus pallidus).

    Findings:

    • Delayed-onset, progressive neurological disorder following strangulation.

    Related Experiment Videos

  • Specific neuropathological findings of bilateral basal ganglia lesions with white matter sparing.
  • Normal EEG and preserved mental state in the initial phase despite severe insult.

    • Implications:

    • The clinical course and preceding state do not predict the occurrence of delayed neurological disorders.
    • Similar pathological findings suggest a common underlying metabolic derangement, possibly lactic acidosis.
    • Further investigation into biochemical defects is warranted for these cases.