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Organophosphate-induced delayed neuropathy: case report.

Luiz Felipe R Vasconcellos1, Ana Cláudia Leite, Osvaldo J M Nascimento

  • 1Institute of Neurology Deolindo Couto, Federal University of Rio de Janeiro, Rio de Janeiro, RJ, Brasil. luizfrv@ig.com.br

Arquivos De Neuro-Psiquiatria
|February 4, 2003
PubMed
Summary

Organophosphate induced delayed neuropathy (OPIDN) is a rare condition following organophosphate ingestion. This case highlights both peripheral and central nervous system involvement, suggesting OPIDN as the more accurate term.

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Area of Science:

  • Neuroscience
  • Toxicology
  • Clinical Neurology

Background:

  • Organophosphate induced delayed neuropathy (OPIDN) is an uncommon neurological disorder.
  • It typically arises after significant organophosphate ingestion, affecting cholinergic receptors.

Observation:

  • A 39-year-old female presented with hyperesthesia and lower limb paresis 14 days post-ingestion of a Dichlorvos-based insecticide.
  • Clinical symptoms included distal paresis and sensory disturbances.

Findings:

  • Electrodiagnostic studies revealed a motor axonal neuropathy.
  • The patient later exhibited pyramidal tract dysfunction in the upper limbs, indicating central nervous system involvement.

Implications:

  • The involvement of both peripheral and central nervous systems supports the designation of organophosphate induced delayed neuropathy (OPIDN).

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  • This case underscores the importance of considering central nervous system effects in organophosphate toxicity.