ARFs

Same author

Lipid transfer protein ORP3 mediates lysosomal repair via LC3B and ubiquitin-TAK1-p38 signaling.

bioRxiv : the preprint server for biology·2026

Same author

Loss of ARF5 impairs recovery after lysosomal damage.

Frontiers in molecular biosciences·2025

Same author

<i>Salmonella</i>-induced cholesterol accumulation in infected macrophages suppresses autophagy via mTORC1 activation.

Molecular biology of the cell·2024

Same author

Quantitation of RhoA activation: differential binding to downstream effectors.

Small GTPases·2022

Same author

The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic.

Molecular biology of the cell·2022

Same author

The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic.

Molecular biology of the cell·2021

Same journal

A global response contributes to tissue size robustness upon local induction of apoptosis.

Current biology : CB·2026

Same journal

Prebilaterian origin of monoaminergic signaling.

Current biology : CB·2026

Same journal

CLASP-dependent microtubule stabilization generates microtubule-based protrusive forces during Drosophila epithelial morphogenesis.

Current biology : CB·2026

Same journal

Pigeons make slow, divergent eye movements during flight and large, convergent eye movements when landing.

Current biology : CB·2026

Same journal

Temperature signals drive grass secondary cell wall thickening.

Current biology : CB·2026

Same journal

Neuronal RNAi and oxygen-sensing circuit shape germline resilience to heat stress.

Current biology : CB·2026

Related Experiment Videos

Related Concept Videos

Lipid transfer protein ORP3 mediates lysosomal repair via LC3B and ubiquitin-TAK1-p38 signaling.

Loss of ARF5 impairs recovery after lysosomal damage.

<i>Salmonella</i>-induced cholesterol accumulation in infected macrophages suppresses autophagy via mTORC1 activation.

Quantitation of RhoA activation: differential binding to downstream effectors.

The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic.

The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic.

A global response contributes to tissue size robustness upon local induction of apoptosis.

Prebilaterian origin of monoaminergic signaling.

CLASP-dependent microtubule stabilization generates microtubule-based protrusive forces during Drosophila epithelial morphogenesis.

Pigeons make slow, divergent eye movements during flight and large, convergent eye movements when landing.

Temperature signals drive grass secondary cell wall thickening.

Neuronal RNAi and oxygen-sensing circuit shape germline resilience to heat stress.

Related Experiment Videos

ARFs

Related Concept Videos

Related Articles

Lipid transfer protein ORP3 mediates lysosomal repair via LC3B and ubiquitin-TAK1-p38 signaling.

Loss of ARF5 impairs recovery after lysosomal damage.

<i>Salmonella</i>-induced cholesterol accumulation in infected macrophages suppresses autophagy via mTORC1 activation.

Quantitation of RhoA activation: differential binding to downstream effectors.

The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic.

The ARF GAPs ELMOD1 and ELMOD3 act at the Golgi and cilia to regulate ciliogenesis and ciliary protein traffic.

A global response contributes to tissue size robustness upon local induction of apoptosis.

Prebilaterian origin of monoaminergic signaling.

CLASP-dependent microtubule stabilization generates microtubule-based protrusive forces during Drosophila epithelial morphogenesis.

Pigeons make slow, divergent eye movements during flight and large, convergent eye movements when landing.

Temperature signals drive grass secondary cell wall thickening.

Neuronal RNAi and oxygen-sensing circuit shape germline resilience to heat stress.