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Immunoactive Properties of Cerebrolysin

Nataliya Yu. Sotnikova1, Olga A. Gromova, Elena A. Novikova

  • 1Ivanovo State Medical Academy, Ivanovo, Russia.

Russian Journal of Immunology : RJI : Official Journal of Russian Society of Immunology
|April 11, 2003
PubMed

Insights

Cerebrolysin treatment in children with minimal cerebral dysfunction improved immune status by increasing CD19(+) cells and normalizing immunoglobulin levels. T helper cell activation was also observed.

Area of Science:

  • Immunology
  • Pediatrics
  • Neurology

Background:

  • Minimal cerebral dysfunction (MCD) in children can be associated with immune system dysregulation.
  • Understanding the impact of therapeutic interventions on immune markers in MCD is crucial.

Purpose of the Study:

  • To investigate the effects of cerebrolysin on the immune status of children aged 3-8 years with MCD.
  • To evaluate changes in lymphocyte subsets, immunoglobulin levels, and activation markers post-treatment.

Main Methods:

  • Children with MCD received intramuscular cerebrolysin (1 ml/10 kg) for one month.
  • Immune status was assessed before and after treatment, analyzing CD19(+), CD4(+), CD16(+) (NK) cells, serum IgG, IgA, and lymphocyte activation markers (CD25, HLA DR, CD95).
  • In vitro studies examined T helper cell activation.

Main Results:

  • Cerebrolysin administration led to an increase in CD19(+) cells and normalization of serum IgG and IgA levels.
  • A rise in CD4(+) lymphocytes and normalization of CD16(+) (NK) cells were observed.
  • Lymphocyte activation markers (CD25, HLA DR) normalized to control group levels, with no change in CD95 expression.
  • In vitro analysis indicated cerebrolysin primarily activates T helper cells.

Conclusions:

  • Cerebrolysin therapy positively modulates the immune status in children with minimal cerebral dysfunction.
  • The treatment enhances B cell counts, normalizes immunoglobulin levels, and improves T cell subsets and NK cell counts.
  • Cerebrolysin demonstrates immunomodulatory effects, particularly on T helper cells, suggesting a potential therapeutic benefit in MCD.

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