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Polymorphisms G691S/S904S of RET as genetic modifiers of MEN 2A

Mercedes Robledo1, Laura Gil, Marina Pollán

  • 1Centro Nacional de Epidemiología, Instituto de Salud Carlos III; Centro Nacional de Investigaciones Oncológicas, Madrid, Spain.

Cancer Research
|April 19, 2003
PubMed

Insights

Genetic variations in the RET proto-oncogene, specifically G691S and S904S polymorphisms, influence the age of diagnosis for Multiple Endocrine Neoplasia type 2A (MEN 2A). These RET variants may also act as low-penetrance risk factors for sporadic thyroid carcinoma.

Area of Science:

  • Genetics
  • Medical Genetics
  • Oncology

Background:

  • Multiple Endocrine Neoplasia type 2A (MEN 2A) is linked to RET proto-oncogene mutations.
  • Identifying susceptibility polymorphisms is crucial for understanding complex human diseases.
  • RET gene variations can influence disease phenotypes and age of onset.

Purpose of the Study:

  • To investigate the role of RET polymorphisms G691S and S904S in MEN 2A.
  • To determine if these polymorphisms affect the age at diagnosis for MEN 2A.
  • To assess the potential of these RET variants as risk factors for sporadic thyroid carcinoma.

Main Methods:

  • Analysis of RET polymorphisms G691S and S904S in 198 individuals from Spanish MEN 2A families.
  • Comparison with a control population of 653 healthy individuals.
  • Statistical analysis including Fisher's exact test and assessment of age at diagnosis and prevalence in sporadic thyroid carcinoma.

Main Results:

  • Strong cosegregation observed between G691S and S904S RET polymorphisms (P < 0.001).
  • Individuals homozygous for these polymorphisms were diagnosed with MEN 2A approximately 10 years earlier (P = 0.037).
  • Homozygous status for these polymorphisms was more prevalent in sporadic thyroid carcinoma patients (OR = 2.36).

Conclusions:

  • The G691S and S904S variants of RET may act as modifier genes influencing the age of onset in MEN 2A.
  • These RET polymorphisms could represent low-penetrance risk factors for sporadic thyroid carcinoma.
  • Further research is warranted to elucidate the precise role of these variants in disease development.

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