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High frequency of human herpesvirus 6 DNA in multiple sclerosis plaques isolated by laser microdissection

Claudio Cermelli1, Rossana Berti, Samantha S Soldan

  • 1Neuroimmunology Branch, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, Maryland 20892, USA.

Insights

Human herpesvirus 6 (HHV-6) DNA was more frequent in multiple sclerosis (MS) plaques than other brain tissues. This suggests HHV-6 may contribute to MS development.

Area of Science:

  • Neurovirology
  • Neuropathology
  • Molecular Biology

Background:

  • Multiple sclerosis (MS) is a chronic inflammatory disease of the central nervous system.
  • The role of viral infections, particularly human herpesvirus 6 (HHV-6), in MS pathogenesis remains under investigation.

Purpose of the Study:

  • To investigate the presence and frequency of HHV-6 DNA in brain autopsy samples from individuals with MS and control groups.
  • To determine if HHV-6 DNA is more prevalent in MS lesions compared to normal-appearing white matter or control brain tissue.

Main Methods:

  • Laser microdissection was used to isolate specific brain tissue areas.
  • DNA extraction followed by nested polymerase chain reaction (PCR) was employed to detect HHV-6 genomic sequences.
  • Samples included MS plaques, normal-appearing white matter (NAWM) from MS patients, non-MS neurological disease controls, and healthy controls.

Main Results:

  • HHV-6 DNA was detected in 15.9% of NAWM samples and 57.8% of MS plaques from patients with MS.
  • The frequency of HHV-6 DNA in MS plaques was significantly higher than in NAWM (P<.0005).
  • HHV-6 DNA detection rates in non-MS neurological disease and healthy control groups were 21.7% and 26.8%, respectively, with no significant difference across sample types overall, except for MS plaques.

Conclusions:

  • HHV-6 DNA is significantly more prevalent in multiple sclerosis plaques.
  • These findings suggest a potential role for HHV-6 in the pathogenesis of multiple sclerosis.
  • Further research is warranted to elucidate the mechanisms by which HHV-6 may contribute to MS.

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