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Mild rhabdomyolysis in a child with fever and "hematuria"
Velibor Tasic1, Vladimir Avramoski, Petar Korneti
1Department of Pediatric Nephrology, Clinic for Children's Diseases, Medical School Skopje, 17 Vodnjanska, 1000, Skopje, Macedonia. vtasic@freemail.com.mk
Insights
This case study shows a 3-year-old girl with mild rhabdomyolysis (muscle breakdown) who recovered fully with outpatient treatment. Early diagnosis and management led to a favorable outcome, preventing recurrence.
Area of Science:
- Pediatric Medicine
- Nephrology
- Biochemistry
Background:
- Rhabdomyolysis is a serious condition involving muscle cell breakdown and release of myoglobin into the plasma.
- Prompt diagnosis and management are crucial for favorable outcomes.
Observation:
- A 3-year-old girl presented with high fever, anorexia, thigh pain, and dark-red urine.
- Urinalysis showed myoglobinuria without red blood cells, confirmed by elevated serum creatine kinase.
Findings:
- The patient was diagnosed with mild rhabdomyolysis.
- Outpatient treatment resulted in complete clinical and biochemical recovery.
- No recurrence of myoglobinuria was observed during a 2-year follow-up period.
Implications:
- Mild rhabdomyolysis in children can be managed effectively on an outpatient basis.
- This case highlights the importance of recognizing and treating pediatric rhabdomyolysis promptly.
- Successful management can lead to full recovery and prevent long-term complications.
Abstract:
Rhabdomyolysis represents a life-threatening condition, which results in release of cellular contents (myoglobin, enzymes, and electrolytes) into the plasma. We report a pediatric patient with mild rhabdomyolysis who had a favorable outcome. A 3-year-old girl had been ill for 2 days with high fever, anorexia, pain in both thighs, and passage of dark-red urine. Myoglobinuria was demonstrated by a "blood"-positive dipstick in the absence of red blood cells in the urinary sediment. Diagnosis was confirmed by the presence of a high serum creatine kinase activity. The child was treated on an outpatient basis and has shown full clinical and biochemical recovery. There has been no recurrence of myoglobinuria during the 2-year follow-up.
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