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Increase in plasma matrix metalloproteinase-9 in acute stroke patients with thrombolysis failure

Ji Hoe Heo1, Seo Hyun Kim, Kyung Yul Lee

  • 1Department of Neurology, Brain Korea 21 Project for Medical Science, Yonsei University College of Medicine, Seoul, Korea. jhheo@yumc.yonsei.ac.kr

Stroke
|May 17, 2003
PubMed
Abstract

Insights

Matrix metalloproteinases (MMPs) may hinder clot breakdown. Higher levels of MMP-9 were found in stroke patients resistant to thrombolytic therapy, suggesting a role in treatment failure.

Area of Science:

  • Biochemistry
  • Neurology
  • Cardiovascular Research

Background:

  • Platelet-rich thrombi present resistance to thrombolytic treatments.
  • Matrix metalloproteinases (MMPs) are implicated in platelet aggregation.
  • MMPs may contribute to thrombolysis failure in stroke patients.

Purpose of the Study:

  • To investigate the role of MMP-2 and MMP-9 in stroke patients undergoing thrombolytic therapy.
  • To determine if MMP levels correlate with thrombolysis resistance.

Main Methods:

  • Plasma samples analyzed from 23 stroke patients and 47 healthy volunteers.
  • Enzyme-linked immunosorbent assays (ELISAs) and zymography used to quantify MMP-2 and MMP-9.
  • Comparison of MMP levels in patients with successful versus failed recanalization.

Main Results:

  • Arterial recanalization was achieved in 15 out of 23 stroke patients.
  • Significantly higher plasma levels of MMP-9 were observed in patients with non-recanalized arteries.
  • No significant difference in MMP-2 levels was noted between groups.

Conclusions:

  • Elevated MMP-9 plasma levels are associated with thrombolysis resistance in stroke.
  • MMP-9 may play a role in the formation of thrombi resistant to pharmacological treatment.
  • Further research is warranted to explore MMP-9 as a therapeutic target.

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