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Characterization of the mupirocin biosynthesis gene cluster from Pseudomonas fluorescens NCIMB 10586

A Kassem El-Sayed1, Joanne Hothersall, Sian M Cooper

  • 1School of Biosciences, University of Birmingham, Edgbaston, Birmingham B15 2TT, United Kingdom.

Chemistry & Biology
|May 29, 2003
PubMed

Insights

Mupirocin, an antibiotic from Pseudomonas fluorescens, inhibits bacterial isoleucyl-tRNA synthase. Its 74 kb biosynthesis gene cluster was sequenced, revealing key genes for producing this important Staphylococcus aureus treatment.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Biochemistry

Background:

  • Mupirocin (pseudomonic acid) is a polyketide antibiotic effective against Staphylococcus aureus, including MRSA.
  • It functions by competitively inhibiting bacterial isoleucyl-tRNA synthase.

Purpose of the Study:

  • To elucidate the genetic basis of mupirocin biosynthesis.
  • To identify the enzymatic functions within the mupirocin gene cluster.

Main Methods:

  • Sequencing of the 74 kb mupirocin biosynthesis gene cluster.
  • Identification of open reading frames (ORFs) and their putative enzymatic functions.
  • Gene knockout and complementation experiments to validate gene functions.

Main Results:

  • The mupirocin cluster comprises six large ORFs (mmpA-F) and individual genes (mupA-X, macpA-E).
  • ORF domains resemble polyketide synthase and fatty acid synthase type I systems, with some genes showing type II system similarity.
  • Gene knockout studies confirmed the essentiality of specific regions for mupirocin production.

Conclusions:

  • A detailed model for mupirocin biosynthesis has been proposed based on sequence and biochemical data.
  • Understanding the biosynthesis pathway provides insights into antibiotic production and potential engineering strategies.

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