[Mutations in the dihydropyrimidine dehydrogenase gene and their role in 5-fluororuracil intolerance]

E Gross1, K Seck, M Kiechle

  • 1Frauenklinik und Poliklinik rechts der Isar, Technische Universität München, Munich. eva.gross@lrz.tum.de

Zentralblatt Fur Gynakologie
|June 25, 2003
PubMed

Insights

Genetic testing for DPYD gene mutations can identify cancer patients at risk for severe side effects from 5-fluorouracil (5-FU) chemotherapy due to dihydropyrimidine dehydrogenase (DPD) deficiency.

Area of Science:

  • Pharmacogenomics
  • Molecular Biology
  • Oncology

Context:

  • Severe side effects from 5-fluorouracil (5-FU) chemotherapy are linked to reduced dihydropyrimidine dehydrogenase (DPD) activity.
  • Over 20 known mutations in the dihydropyrimidine dehydrogenase gene (DPYD) may cause DPD deficiency, but genotype-phenotype correlations are often unclear.

Purpose:

  • To determine the frequency of DPD defects in the German population.
  • To identify novel DPYD mutations associated with 5-FU toxicity.
  • To develop a sensitive and efficient screening method for DPYD mutations in cancer patients.

Summary:

  • A German population study investigated DPYD gene mutations using denaturing HPLC (DHPLC).
  • DHPLC proved to be a reliable, cost-effective, and time-efficient method for analyzing large sample sizes.
  • Further analysis of patients receiving 5-FU will elucidate the molecular basis of DPD deficiency.

Impact:

  • Enables identification of cancer patients at risk for 5-FU-related toxicity.
  • Facilitates personalized chemotherapy regimens, improving patient safety.
  • Contributes to understanding genotype-phenotype relationships in DPYD mutations.

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