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Natural history of asthma

Theresa Guilbert1, Marzena Krawiec

  • 1Division of Pediatric Pulmonary Medicine, Arizona Respiratory Center, University of Arizona, 1501 N. Campbell Avenue, Tucson, AZ 85724, USA. guilbert@resp-sci.arizona.edu

Insights

Childhood asthma can be persistent, leading to lifelong severe symptoms and lung function loss, particularly in those with a family history. New research is needed to understand evolving asthma patterns and genetics in children.

Area of Science:

  • Pediatric respiratory medicine
  • Clinical immunology
  • Epidemiology of chronic diseases

Background:

  • Asthma in children presents variable natural histories, with some experiencing symptom remission while others develop persistent, severe disease.
  • Persistent childhood asthma is often linked to a family history of asthma, increased airway reactivity, and atopy.
  • Longitudinal studies have improved understanding of asthma's natural history in children, but critical questions remain.

Purpose of the Study:

  • To review current understanding of childhood asthma natural history based on longitudinal studies.
  • To highlight the need for new cohort studies investigating potential changes in asthma incidence, severity, and pathophysiology in recent birth cohorts.
  • To emphasize the importance of genetic investigations for identifying distinct asthma phenotypes and guiding future therapeutic interventions.

Main Methods:

  • Review of findings from existing longitudinal studies on childhood asthma.
  • Discussion of epidemiological trends in asthma incidence and severity.
  • Call for new prospective cohort studies and genetic research.

Main Results:

  • Some children experience asthma remission, while others develop persistent disease with significant morbidity.
  • Children with persistent asthma often exhibit early signs of airway hyperresponsiveness and atopy, alongside a positive family history.
  • Increasing asthma incidence and severity suggest potential shifts in disease pathophysiology or environmental influences in recent generations.

Conclusions:

  • Further longitudinal and genetic studies are crucial to understand the evolving landscape of childhood asthma.
  • Identifying distinct asthma phenotypes early in life could enable personalized therapeutic strategies to mitigate symptom severity and lung function decline.
  • Understanding the genetic basis of asthma is key to elucidating its complex phenotypic expressions.

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