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Mannose receptor contribution to Candida albicans phagocytosis by murine E-clone J774 macrophages

Isabelle Porcaro1, Michel Vidal, Sylvie Jouvert

  • 1Laboratoire d'Immunologie et de Parasitologie EA 2413, Université Montpellier I, France. UMR CNRS 5539, Université Montpellier II, France.

Insights

Mannose receptor (MR) on macrophages binds Candida albicans, promoting yeast ingestion. Phagocytosis reduces MR endocytic activity but not receptor degradation.

Area of Science:

  • Immunology
  • Cell Biology
  • Microbiology

Background:

  • Yeast mannoproteins interact with macrophage mannose receptors (MR) for opsonin-independent phagocytosis.
  • Mannose receptor (MR) mediates endocytosis of soluble ligands via clathrin-coated pits.

Purpose of the Study:

  • Investigate the interaction between MR and Candida albicans.
  • Elucidate the consequences of this interaction on MR trafficking in macrophages.

Main Methods:

  • Utilized pull-down assays with mannosylated Sepharose beads and E-clone macrophage lysates.
  • Employed glutaraldehyde-fixed, heat-killed (HK) Candida albicans for binding studies.
  • Assessed MR endocytic activity and receptor degradation in E-clone macrophages.

Main Results:

  • Solubilized MR binding to mannosylated particles mirrored cell-surface MR activity.
  • MR on E-clone macrophages facilitated phagocytosis of unopsonized, HK C. albicans.
  • Yeast phagocytosis led to decreased MR endocytic activity without receptor degradation.

Conclusions:

  • Macrophage mannose receptor (MR) plays a role in the direct recognition and phagocytosis of Candida albicans.
  • MR trafficking is modulated by yeast phagocytosis, with reduced endocytic activity observed.
  • The mannose receptor (MR) is not degraded during the phagocytosis of Candida albicans within the studied timeframe.

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