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Prickle and Strabismus form a functional complex to generate a correct axis during planar cell polarity signaling

Andreas Jenny1, Rachel S Darken, Paul A Wilson

  • 1Mount Sinai School of Medicine, Brookdale Department of Molecular, Cellular and Developmental Biology, 1 Gustave L.Levy Place, New York, NY 10029, USA.

The EMBO Journal
|August 28, 2003
PubMed

Insights

Planar cell polarity (PCP) genes prickle (Pk) and strabismus (Stbm) physically interact and localize to the cell membrane. This Stbm/Pk complex modulates Frizzled/Dishevelled signaling, crucial for symmetry breaking in Drosophila eye development.

Area of Science:

  • Developmental Biology
  • Cell Signaling
  • Genetics

Background:

  • Planar cell polarity (PCP) pathways establish tissue polarity.
  • Frizzled (Fz) signaling is central to PCP.
  • Prickle (Pk) and strabismus (Stbm) are PCP genes proposed to antagonize Fz signaling.

Purpose of the Study:

  • To investigate the interaction and cellular localization of Pk and Stbm.
  • To elucidate the role of the Pk/Stbm complex in Frizzled/Dishevelled (Dsh) signaling.
  • To understand the mechanism by which Pk and Stbm contribute to symmetry breaking in Drosophila eye development.

Main Methods:

  • Co-immunoprecipitation to assess physical interactions between Stbm and Pk.
  • Confocal microscopy to visualize protein localization at the cell membrane.
  • Analysis of gene function in Drosophila eye development.

Main Results:

  • Stbm and Pk interact physically and co-localize at the cell membrane.
  • Stbm recruits Pk to the membrane, affecting Stbm localization and clustering.
  • The Stbm/Pk complex appears to modulate Fz/Dsh pathway activity.

Conclusions:

  • Pk and Stbm function within the same cell to regulate PCP.
  • The Stbm/Pk complex plays a key role in modulating Fz/Dsh signaling.
  • This interaction is critical for the symmetry-breaking step in Drosophila polarity signaling.

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