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The novel conjugative transposon tn1207.3 carries the macrolide efflux gene mef(A) in Streptococcus pyogenes

Maria Santagati1, Francesco Iannelli, Carmela Cascone

  • 1Department of Microbiology, University of Catania, 95124 Catania, Italy. santagatim@tiscali.it

Microbial Drug Resistance (Larchmont, N.Y.)
|September 10, 2003
PubMed

Insights

A mobile genetic element, Tn1207.3, carrying the macrolide efflux gene mef(A) was identified in Streptococcus pyogenes. This conjugative transposon can transfer the mef(A) gene between streptococcal species, contributing to macrolide resistance.

Area of Science:

  • Microbiology
  • Genetics
  • Molecular Biology

Background:

  • Macrolide resistance is a growing concern in bacterial infections.
  • The mef(A) gene confers resistance to macrolide antibiotics.
  • Mobile genetic elements play a crucial role in the dissemination of antibiotic resistance genes.

Purpose of the Study:

  • To characterize the genetic element carrying the mef(A) gene in Streptococcus pyogenes.
  • To investigate the transferability and insertion characteristics of this element in other streptococcal species.

Main Methods:

  • Pulsed-field gel electrophoresis (PFGE) to determine element size.
  • DNA sequencing to analyze genetic element structure and homology.
  • Conjugation experiments to assess transferability between bacterial species.

Main Results:

  • A 52-kb conjugative transposon, named Tn1207.3, carrying the mef(A) gene was identified in S. pyogenes.
  • Tn1207.3 shares homology with the pneumococcal Tn1207.1 element.
  • Tn1207.3 was successfully transferred to Streptococcus pneumoniae and Streptococcus gordonii, inserting at a specific chromosomal site.
  • Tn1207.1 appears to be a defective part of a larger conjugative element.

Conclusions:

  • Tn1207.3 is a mobile genetic element responsible for the spread of the mef(A) gene in S. pyogenes.
  • The conjugative nature of Tn1207.3 facilitates the horizontal transfer of macrolide resistance among streptococci.
  • Understanding the mobility of such elements is critical for combating antibiotic resistance.

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